Efecto del cigarrillo sobre la mucosa oral
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2018-05
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Universidad de Guayaquil. Facultad Piloto de Odontologìa
Resumen
La mucosa bucal es susceptible a diferentes cambios que son tanto intrínsecos como extrínsecos, los hábitos pueden provocar diferentes cambios.
El hábito de fumar es uno de los que se han estudiado en la actualidad por lo cual es de importancia resaltar que es deber de los odontólogos y todos quienes trabajamos en temas de salud, fomentar y colaborar en la entrega de herramientas para apoyar a los pacientes en la difícil misión de abandonar el mal hábito, fumar retarda los procesos fisiológicos de reparación, que es un daño que, lamentablemente, se acumula Objetivo: Definir, las lesiones de la mucosa bucal relacionadas con el hábito de fumar. Metodología: Descriptivo, Correlacional NO experimental, Métodos: teóricos-prácticos: Resultados: Autores afirman en numerosas publicaciones establecen una clara asociación entre el hábito de fumar y la prevalencia y severidad de las lesiones, estudios anteriores reportaban más inflamación gingival y placa bacteriana en fumadores, sin embargo estudios recientes establecen que cuando el nivel de placa se ha controlado los fumadores muestran menor inflamación y sangrado gingival que los no fumadores, indicando una respuesta inflamatoria deprimida. Las lesiones de la mucosa bucal se relacionan significativamente: hiperqueratosis, hiperparaqueratosis, hiperplasia epitelial, vascularización aumentada a nivel sub epitelial dentro del tejido conectivo, promoción de la respuesta inflamatoria evidenciada en un incremento de la cantidad de células por campo. Está claro que los productos del tabaco producen un efecto vasoconstrictor en los tejidos, la nicotina es capaz de inhibir la producción de citoquinas antiinflamatorias y quimiotácticas en el endotelio deteriorando el mecanismo inmunológico de defensa contra los cambios epiteliales de la mucosa bucal y finalmente permitiendo a las células neoplásicas epiteliales burlar la vigilancia inmunológica e invadir los tejidos subyacentes.
The buccal mucosa is susceptible to different changes that are both intrinsic and extrinsic, habits can cause different changes. The habit of smoking is one of those that have been studied today so it is important to highlight that it is the duty of dentists and all those who work on health issues, encourage and collaborate in the delivery of tools to support patients In the difficult mission of giving up the bad habit, smoking slows down the physiological processes of repair, which is a damage that, unfortunately, accumulates Objective: To determine, buccal mucosal lesions related to smoking. Methodology: Descriptive, Correlational NO experiments, Methods: practical theorists: Results: Authors affirm in numerous publications establish a clear association between smoking habit and the prevalence and severity of periodontal disease, previous studies reported more gingival inflammation and bacterial plaque in smokers However, recent studies establish that when the level of plaque has been controlled, smokers show less inflammation and gingival bleeding than non-smokers, indicating a depressed inflammatory response. Buccal mucosal lesions are significantly related: hyperkeratosis, hyper parakeratosis, epithelial hyperplasia, increased vascularization at the sub-epithelial level within the connective tissue, promotion of the inflammatory response evidenced by an increase in the number of cells per field. It is clear that tobacco products produce a vasoconstrictor effect in the tissues, nicotine is able to inhibit the production of anti-inflammatory and chemotactic cytokines in the endothelium, deteriorating the immune defense mechanism against the epithelial changes of the buccal mucosa and finally allowing the neoplastic epithelial cells circumvent immune surveillance and invade underlying tissues
The buccal mucosa is susceptible to different changes that are both intrinsic and extrinsic, habits can cause different changes. The habit of smoking is one of those that have been studied today so it is important to highlight that it is the duty of dentists and all those who work on health issues, encourage and collaborate in the delivery of tools to support patients In the difficult mission of giving up the bad habit, smoking slows down the physiological processes of repair, which is a damage that, unfortunately, accumulates Objective: To determine, buccal mucosal lesions related to smoking. Methodology: Descriptive, Correlational NO experiments, Methods: practical theorists: Results: Authors affirm in numerous publications establish a clear association between smoking habit and the prevalence and severity of periodontal disease, previous studies reported more gingival inflammation and bacterial plaque in smokers However, recent studies establish that when the level of plaque has been controlled, smokers show less inflammation and gingival bleeding than non-smokers, indicating a depressed inflammatory response. Buccal mucosal lesions are significantly related: hyperkeratosis, hyper parakeratosis, epithelial hyperplasia, increased vascularization at the sub-epithelial level within the connective tissue, promotion of the inflammatory response evidenced by an increase in the number of cells per field. It is clear that tobacco products produce a vasoconstrictor effect in the tissues, nicotine is able to inhibit the production of anti-inflammatory and chemotactic cytokines in the endothelium, deteriorating the immune defense mechanism against the epithelial changes of the buccal mucosa and finally allowing the neoplastic epithelial cells circumvent immune surveillance and invade underlying tissues
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TRAUMATISMO DE LOS DIENTES, HABITOS BUCALES, SALUD BUCAL